Research has identified that younger than 6 months of age, male, incomplete KD, longer fever duration, higher CRP levels (> 100 mg/l), and lower albumin levels (< 35 g/L) were all independent risk factors for CAA formation [20], thus indicating that both delayed initiation of KD target therapy and hypoalbuminemia, which indicates a relatively poor nutritional status, result in higher incidence rates of CAA complications in patients with acute KD, despite the administration of IVIG therapy. The gene discussed is ALB; the disease is Hypoalbuminemia.