KIT and sarcoma: The putative role of FGFR signaling through FGF2 in KIT-mutant GISTs can be supported also by gene expression data metanalysis, which suggest that GISTs express FGF2 at levels significantly higher than other sarcomas (Figure 3C) and that, among GISTs, KIT-mutant samples exhibit the largest heterogeneity of expression (Figure 3D).