Nucleus-translocated CaMKII isoforms have shown previously and in the present study to regulate cell cycle in irradiated K562 CML [(Heise et al., 2010) and present study], Jurkat T cell leukemia (Klumpp et al., 2016), and glioblastoma cells (Klumpp et al., 2017). Here, CAMK2G is linked to T-cell leukemia.