The 84 different reinterpreted variants in this study represent the distribution of LQTS‐causing genes reported in the literature with KCNQ1, KCNH2, and SCN5A being the main cause, although variants in KCNQ1 were overrepresented with 50% (compared to 30%–35% in the literature; Alders et al., 1993). Here, KCNH2 is linked to familial long QT syndrome.