However, effects on proliferation and viability seemed to be independent from the cleavage and activity of the apoptosis marker PARP-1 and caspases 3, 7, and 8 in breast cancer (23 μM [Loganathan et al., 2013]), pancreas cancer (50 μM [Husain et al., 2011]), and colon cancer cells (100 μM [Campbell et al., 2006]). This evidence concerns the gene PARP1 and pancreatic neoplasm.