AGT and hypertensive disorder: miR‐103a‐3p always serves as an oncogene (Hong et al., 2014; Hu et al., 2018; Xiong et al., 2017) and it is increased in the plasma of hypertension patients (Bacon et al., 2015; Karolina et al., 2012), and it is able to aggravate angiotensin II‐induced renal inflammation and fibrosis (Lu et al., 2019), implying that miR‐103a‐3p might advance the septic liver failure.