For example, miR‐191‐5p attenuates sepsis‐induced kidney injury via binding to oxidative stress‐responsive 1 (Qin, Wang, & Peng, 2019), and miR‐106a enhances sepsis‐mediated acute kidney failure by targeting THBS2 (Shen, Yu, Jing, & Zhang, 2019). The gene discussed is THBS2; the disease is Sepsis.