Additionally, miR‐103a‐3p is upregulated in the plasma of hypertension patients (Karolina et al., 2012) and it is also elevated in the urine of diabetes mellitus patients (Bacon, Engelbrecht, Schmid, Pfeiffer, & Gallagher, 2015), and moreover, miR‐103a‐3p promotes angiotensin II‐induced renal inflammation and fibrosis by modulating SNRK/NF‐κB/p65 signaling (Lu et al., 2019), suggesting that miR‐103a‐3p might also aggravate septic liver injury. Here, AGT is linked to hypertensive disorder.