miR‐103a‐3p always serves as an oncogene (Hong et al., 2014; Hu et al., 2018; Xiong et al., 2017) and it is increased in the plasma of hypertension patients (Bacon et al., 2015; Karolina et al., 2012), and it is able to aggravate angiotensin II‐induced renal inflammation and fibrosis (Lu et al., 2019), implying that miR‐103a‐3p might advance the septic liver failure. Here, AGT is linked to Hypertension.