TP53 and neoplasm: However, the coinstantaneous treatment of tumour cells with PEMF and agonist of A(3) adenosine receptors (A3AR)—2-chloro-N 6-(3-iodobenzyl)adenosine-5′-N-methyl-uronamide (Cl-IB-MECA)—caused a further significant elevation of protein levels of p53, compared with Cl-IB-MECA alone [35].