Collectively, our findings of increased fatty acid transporter expression (CD36, FABP3, and FABP4), enhanced triglyceride accumulation, and concomitant reduction in DHA transport in GDM conditions suggest that the DHA is taken up by the trophoblasts and retained after esterification into triglycerides instead of being transferred to the fetus. The gene discussed is FABP3; the disease is gestational diabetes.