In the case of Duchenne muscular dystrophy (DMD), the dystrophin-deficient mice (mdx mice) have provided a valuable model to study certain common aspects of the disease [1], but the model is unfortunately not informative on patient-specific pathological features that could only be appreciated by using “in dish” disease models from human patient-derived induced pluripotent stem cells (iPSC). The gene discussed is DMD; the disease is Duchenne muscular dystrophy.