Susceptibility to the development of post–COVID‐19 pulmonary fibrosis could also have a genetic component, as variants in numerous genes or their promotors (such as MUC5B and TERT) have been found to predispose to lung fibrosis, both in the context of idiopathic pulmonary fibrosis and in interstitial fibrosis related to hypersensitivity pneumonitis and collagen vascular disease.27, 28, 29, 30. Here, TERT is linked to pulmonary fibrosis.