These included canonical genes associated with Th1 cells (T-bet, IFNγ), a subset highly permissive to HIV infection and stably maintained during antiretroviral therapy [27]; Ox40 and BCL2, which can promote survival of HIV-infected cells [10, 28, 29]; cyclinT1, necessary for efficient elongation of HIV transcripts [30]; metabolic genes (e.g., MTOR) that promote HIV transcription [31]; and CD30 expressed on cells with transcriptionally active HIV [32]. Here, MTOR is linked to HIV infectious disease.