indicated that chemokines (e.g., CCL2, CCL3, CCL4, CCL5, CXCL9, and CXCL10) allow effector T cells to migrate into the TME to mediate tumor destruction, but poor chemokine-mediated trafficking of T cells favors tumor growth.119, 120, 121 Lacking evidence, the source of released chemokines during efferocytosis in tumors remains unclear. The gene discussed is CCL5; the disease is neoplasm.