Additionally, we also analyzed changes in phenotype and cytokine production in healthy moDCs upon exposure to hetelogous T1D-NETs and found out that HD-moDCs expressed significantly higher level of CD86, HLA-DR, PDL1 and produced markedly more proinflammatory cytokines under these conditions (Figures 4G,H). The gene discussed is CD86; the disease is type 1 diabetes mellitus.