The presence of these factors is in agreement with higher capacity of IFNγ-inflamed MSCs to recruit T cells at their proximity and ability to reduce the symptoms of graft-versus-host disease (GVHD) in a mouse model [75], being CCL8 and CXCL9/10 upregulated in the mouse system after inflammation [76]. The gene discussed is CCL8; the disease is graft versus host disease.