AZGP1 and epilepsy: Similarly, in neurons, we found ZAG promoted ketogenesis, and ZAG overexpression alleviated epilepsy/seizure‐induced impairments in neuronal ketogenesis, while knockdown of ZAG suppressed ketogenesis in cultured neurons, similar to the Mg2+‐free ACSF treatment, supporting the role of ZAG in seizure‐induced impairment in ketogenesis.