Studies in cervical cancer have only evaluated changes in the expression of nonphosphorylated HSP27; nevertheless, in HeLa cells, it has been reported that phosphorylated HSP27 is involved in the apoptosis induced by TNF-α mediating its interaction with TAK1 and regulating the posttranslational modifications of TAK1 [44]. Here, HSPB1 is linked to cervical carcinoma.