EGR1 and gastric cancer: In our previous study, we found that EGR1-activated HNF1A-AS1 transcription promoted cell growth and cell cycles in GC, and that it could also increase GC cell migration abilities.14 At present, whether HNF1A-AS1 was dysregulated in GC needs to be addressed, as well as the association between HNF1A-AS1 expression and the clinicopathological parameters of the patients with GC.