Other studies showed that a constitutively active ERBB4 homodimer mutant inhibited colony formation in a pancreatic cancer cell line (Mill et al., 2011), and classified ERBB4 as a potential oncogene, after revealing enhanced anchorage‐independent growth of hPaCaCells by the stimulation of ectopic ERBB4 expression (Mill et al., 2011). Here, ERBB4 is linked to pancreatic neoplasm.