Taken together, ER stress may upregulate GOLGA2P10 through the PERK/ATF4/CHOP pathway, and the elevated GOLGA2P10 may confer tumor cells with resistance to ER stress-induced apoptosis by increasing the protein level of BCL-xL and the phosphorylation of BAD (Fig. 6f). Here, DDIT3 is linked to neoplasm.