CALR and neoplasm: Studies have revealed that complementary immunostimulatory mechanisms were also activated after radiation, including the activation of the stimulator of interferon genes (STING)‐mediated DNA‐sensing pathway, and the upregulation of the MHC‐I and calreticulin expression.[61] These additional mechanisms further activated dendritic cells and enhanced cross‐presentation of tumor antigens, thus promoting antitumor immune responses.