Indeed, while in controls and a group of GD (Group2, n = 3 out of 11, 27%), ILCs could not be observed in tissues, we observed a high proportion of LTi-like ILC3 (CRTH2− C-kit+ NKp44−) among infiltrating ILCs in the tissues of all HT patients and another group of GD patients (Group1, n = 8 out of 11, 83%) (Figure 3C). This evidence concerns the gene PTGDR2 and hematocrit.