In a study of 44 children with TBM-associated hydrocephalus, 11 healthy controls and 9 children with pulmonary TB, neuromarkers of brain damage; S100B, neuron-specific enolase and glial fibrillary acidic protein, were elevated for at least 3 weeks in the TBM group [6]. The gene discussed is GFAP; the disease is meningeal tuberculosis.