KLF4 and neoplasm: One study reported SNHG5 as an oncogene that stimulates cell proliferation and migration, by sponging miR-32 and up-regulation of KLF4 (Krüppel-like factor 4) (87), while another study reported that SNHG5 is a tumor suppressor, which interacts with MTA2 (metastasis associated 1 family member 2), and it impairs the MTA2 translocation from the cytoplasm to the nucleus.