As a result of oxidative stress, tests on mucoepidermoid carcinoma cell lines have shown an increase in the cytoplasmic concentration of HSP60; if paired with a massive release from mitochondrial compartment, the role of HSP60 is pro apoptotic; otherwise, if the mitochondrial release is absent, the chaperone performs an anti-apoptotic action. This evidence concerns the gene HSPD1 and mucoepidermoid carcinoma.