SLC16A3 and Sepsis: The adult sepsis group exhibited indirect interaction between the CD177, GPR84, and TDRD9 hubs whereas the pediatric sepsis group exhibited indirect interaction between CD177, GPR84 and TDRD9; direct interaction between the GPR84 and TDRD9 hubs, indirect interaction between TDRD9 and the FGF13 and PCOLCE2 hubs and also between the PCOLCE2 and SLC16A3 hubs.