QRSL1 and endometriosis: DNA methylation seems to be an integral component of endometriosis, and according to Dyson et al. [48], the GATA family is a regulator of uterine physiology and aberrant DNA methylation in endometriotic cells correlates with a shift in GATA isoform expression that permits GATA6 expression in endometriosis instead of GATA2.