We hypothesize that the increased presence of PRDX1, induced by the altered redox and bioenergetic homeostasis that characterize TNBC cells [41], stabilizes to some extent the redox homeostasis in breast cancer cells, decreases further spontaneous mutagenesis, and slows down its progression into more aggressive forms, and thus it correlates with a more favorable prognosis. This evidence concerns the gene PRDX1 and breast cancer.