In the present study, we show that PT specimens harbored typical prostate adenocarcinoma features including a luminal morphology, epithelial markers, PSA and AR expression together with clonal TMPRSS2-ERG fusion and TP53 mutation, and two subclonal TP53 losses (17p12-13 and 17p12-tel). This evidence concerns the gene TMPRSS2 and prostate adenocarcinoma.