It was reported that a potential pathologic p.G76S heterozygous mutation on the TNFRSF13B gene which identified by whole‐exome sequencing might upregulate cytokine‐cytokine receptor interaction signaling pathway and increase serum TNFα, IL‐17α, IFNγ and BAFF levels in immune thrombocytopenia patients.35 The gene discussed is IL17A; the disease is autoimmune thrombocytopenic purpura.