FTO and acute myeloid leukemia: 3-deazaadenosine (DAA) interrupts METTL3/14 and inhibits the decoration of m6A by obstructing SAH hydrolase [165], SPI1 is considered a potential target for AML because of inhibition of METTL14 [112], and meclofenamic acid (MA), a non-steroidal anti-inflammatory drug, silences FTO by competing for binding sites [166].