Fusion breakpoints occurred within intron 3 of PML (like those observed in PML-RARA bcr3 fusions) and the 5’ untranslated region (UTR) of RARG. The unique patient with clinical features of classical APL received ATRA treatment for 9 days, but did not exhibit a differentiation [90]. The gene discussed is PML; the disease is acute promyelocytic leukemia.