Since intrauterine hyperglycemia and neonatal hypoglycemia have been shown to be a risk factor for ASD79, further investigations in cohorts with information on neonatal glycemia, maternal lifestyle or history of type I diabetes would be needed to test the hypothesis of a correlation between altered PTPRN2 expression in ASD and abnormalities in glucose or in prediabetes markers. The gene discussed is PTPRN2; the disease is type 1 diabetes mellitus.