In our study, the results showed that MI induced increased myocardial cell apoptosis accompanied with an increase in Bax protein expression and a reduction in Bcl2 protein expression, YXK treatment significantly inhibited myocardial cell apoptosis after MI, suggesting that the improvement of cardiac function by YXK was at least partially due to reduced myocardial cell apoptosis. This evidence concerns the gene BAX and myocardial infarction.