TRPV1 and atherosclerosis: Ching’s study showed that after excitation of TRPV1 by evodiamine, the AMPK and eNOS phosphorylation in aortic endothelial cells increased, which promoted the formation of the TRPV1- eNOS complex, increased the production of NO, promoted the formation of new blood vessels, and delayed the generation of atherosclerosis in mice [38].