ARID1A and granular cell tumor: In GCTs, ARID1A mutations are extremely rare, so we asked, if a romidepsin-mediated ARID1A downregulation, a CRISPR/Cas9-mediated ARID1A deficiency, or a pharmacological inhibition of ARID1A might phenocopy these effects of the ARID1A mutation and sensitize GCT cells to the various previously mentioned inhibitors.