Later on, other studies supported the induction of a β-catenin-IRF3-p300/CBP complex upon infection with Sendai virus (SeV) and influenza A virus H1N1 [79,80,83] that acts in parallel to the RLR-TBK1-IRF3 pathway to fine-tune the antiviral innate immune response. The gene discussed is IRF3; the disease is infection.