OLR1 and preeclampsia: This suggests that having both increased levels of circulating oxLDL and STBEVs could worsen the effect of either of these factors alone and, together with our endothelium-dependent vasodilation data, this is in line with the general hypothesis that vascular dysfunction in women with preeclampsia (i.e. conditions of increased LOX-1 expression) is multifactorial and heterogenous3.