Using selective inhibitors of the HSP90 chaperone activity or techniques with HSP90 knockdown, it was shown that intracellular HSP90 can be conducive to EMT in carcinomas of different localization [65,66,67,68,69,70,71], while the mechanisms of such HSP90-dependent EMT may vary in different cases. This evidence concerns the gene HSP90AB1 and carcinoma.