This is supported by a study using two murine models of kidney fibrosis (the unilateral ureteric obstruction model and the ischemia-reperfusion renal fibrosis model) crossed with recombinant mice in which the renal epithelial and stromal cells were genetically labeled: this study showed that during nephrogenesis, FoxD1+ mesenchymal cells become adult CD73+ PDGFRβ+ α-SMA- pericytes, and that in fibrogenic conditions, these pericytes start expressing α-SMA. The gene discussed is ACTA1; the disease is Ureteral obstruction.