Nonetheless, since the levels of CDK6, and the prosurvival protein BCL2 were modulated upon BORA silencing and inhibitors of these pathways are FDA-approved drugs for breast cancer and leukemia [44,45], we sought to ascertain whether the combination of these inhibitors could mimic the BORA depletion phenotype and thus could result in beneficial therapeutic effects for OC patients. The gene discussed is BCL2; the disease is leukemia.