CT inhibited p-STAT5 and p-STAT3, effectively blocked IL-6–mediated STAT3 activation, and reversed chronic myeloid leukemia (CML) fusion gene (BCR-ABL) kinase-independent drug resistance and inhibited key cell coproliferation and drug resistance pathway of K562/ADR in CML (Dong B. et al., 2018). This evidence concerns the gene ABL1 and chronic myelogenous leukemia, BCR-ABL1 positive.