OXT and obesity due to melanocortin 4 receptor deficiency: A number of general biological processes have been proposed to underpin the prenatal adaptations that heighten offspring obesity risk, including oxidative stress; epigenetic processes; glucocorticoid effects; as well as the actions of neuroactive steroids, somatolactogenes, and related peptides, such as insulin-like growth factor (IGF-1) and oxytocin [13,14,15].