miR-210 is well-recognized as being highly upregulated in hypoxia in a variety of solid tumors including glioblastomas (42, 50–58), a finding which has been further confirmed mechanistically by both the identification of a hypoxia response element (HRE) in its promoter (42) and the close correlation of miR-210 expression with vascular endothelial growth factor (VEGF) expression (53). The gene discussed is VEGFA; the disease is glioblastoma.