Treatment with BAW decreased the infiltration of inflammatory cells, bronchial thickness, serum concentration of OVA-specific IgE and expression of Th2 cytokines, while a significant recovery was observed on the goblet cell hyperplasia, MMP-9 expression, VEGF signaling pathway, acetylcholine esterase (AChE) activity and muscarinic acetylcholine receptor (mAChR) M3 downstream signaling pathway in an ovalbumin (OVA)-induced asthma model after treatment with BAW [15]. Here, ACHE is linked to asthma.