RSV also regulates fibroblast growth factor 2 (FGF2), a paracrine molecule that contributes to cardiac fibrosis by binding to its receptors, such as FGFR1 and heparan sulfate proteoglycans (HSPGs), on the surface of mCFs; from there, it subsequently exerts its antifibrotic ability in diabetes [138–140]. Here, FGF2 is linked to diabetes mellitus.