STAT3 and gastric cancer: Since activation of STAT3 or P38 signaling pathways have been described as mediators of survival in response to cisplatin, we evaluated the levels of pSTAT3 and/or pP38 in the MKN74 isogenic model and in the GC cells that endogenously overexpress CD44v6, GP202 and MKN45 (Figure 4C–E).