CFIm25 knockdown leads to at least 1,450 genes with shortened 3′UTRs and marked increases in the expression of several oncogenes in GBM, including cyclin D1 (CCND1), glutaminase (GLS) and methyl-CpG-binding protein 2 (MECP2), thereby enhancing their tumorigenesis [147]. This evidence concerns the gene GLS and glioblastoma.