Since Foxf1 has been reported to inhibit pulmonary fibrosis by preventing CDH2 to CDH11 cadherin switch in myofibroblasts during pulmonary fibrosis [22], we proposed that Kl be very likely to repress the TGF-β-induced migration of pulmonary fibroblasts via enhancing the expression of Foxf1. As hypothesized, rKL supplementation restored Foxf1 mRNA and protein levels in both PCLSs treated with FC (Figure 6D and 6E) and pulmonary fibroblasts incubated with TGF-β (Figure 6F and 6G). The gene discussed is KL; the disease is pulmonary fibrosis.