Indeed, we found that they not only had increased expression in most of the 16 tested cancer types, but they also had similar correlation pattern with immune infiltration subtypes, where they were highly expressed in immune subtypes (i.e., C1, C2, and C6) with high proliferation rate and enriched with IFN-r infiltrates, indicating a tumour promoter role. The gene discussed is IFNAR2; the disease is neoplasm.