It has been shown that the activation of the Akt/mTOR pathway by the long noncoding RNAs OECC [23] and MetaLnc9 [24] and the transmembrane 7 superfamily member 4 [25] promotes cancer metastasis; conversely, the suppression of the Akt/mTOR pathway in the presence of the ferulic acid derivative FXS-3 [26], cardamonin [27] and microRNA-520a-3p [28] inhibits cancer metastatic potential. This evidence concerns the gene AKT1 and cancer.